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For Lyme disease and cancer specifically, the oxidative stress driving cellular damage can be directly counteracted at these concentrations in ways that feel more like supporting the body's own defenses than imposing a foreign treatment

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Its anti-glycation potential has also been confirmed in models of premature aging, such as D-Gal-induced RBC damage, where quercetin fully prevented HbA1c formation, suggesting that it interferes with early glycation events at the Hb level [223]